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The mechanics of addiction - social media vs drugs

Apr 12, 2024, Update: Apr 12, 2024, author: Everyoung.com
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"This article explores the roles of serotonin and dopamine, two vital neurotransmitters, and their impact on the brain's reward circuits and addiction mechanisms. Given the pivotal role that overcoming addictions plays in the Everyoung Health Method, we aim to shed light on the foundational principles that underpin this approach."

1. Serotonin


The Modulator of Neural Networks

Serotonin, scientifically known as 5-hydroxytryptamine (5-HT), is a multifaceted neurotransmitter that plays a pivotal role in the modulation of numerous neural networks. It is synthesized from the amino acid tryptophan in a two-step process involving the enzymes tryptophan hydroxylase and aromatic amino acid decarboxylase. Beyond its well-known functions in mood regulation, serotonin influences various physiological processes, including sleep, appetite, and pain perception. It operates across the brain, engaging with multiple serotonin receptors, categorized into seven leading families (5-HT1 to 5-HT7), each with distinct subtypes and functions.

Serotonin and addictions

In the context of addiction, serotonin's role is nuanced. It modulates the reward system indirectly, influencing the salience of rewards and the individual's impulsivity. Variations in serotonin levels and receptor function have been associated with different addictive behaviors. For example, decreased serotonin activity can lead to an increased vulnerability to addiction by reducing the individual's ability to delay gratification and increasing impulsivity.

2. Dopamine


The Driver of Reward and Motivation

Dopamine, on the other hand, is often heralded as the "pleasure molecule," a somewhat oversimplified characterization of its complex role in the brain's reward system. It is synthesized from the amino acid tyrosine in a two-step process catalyzed by tyrosine hydroxylase and DOPA decarboxylase. Dopamine operates through five main receptor types, D1 through D5, which are divided into two families based on their action mechanisms: D1-like receptors (D1 and D5) that stimulate adenylate cyclase, and D2-like receptors (D2, D3, and D4) that inhibit adenylate cyclase.

Dopamine is critically involved in the brain's reward circuitry, particularly within the mesolimbic pathway, which extends from the ventral tegmental area (VTA) to the nucleus accumbens, a key area for reward processing. When an action leads to a reward, dopamine neurons in the VTA are activated, releasing dopamine in the nucleus accumbens. This dopamine signal encodes the prediction and experience of reward, reinforcing the behavior that led to the reward and making it more likely to occur in the future.

3. Interplay in Addiction


The interplay between serotonin and dopamine is critical in addiction. Addictive substances and behaviors can hijack the brain's reward system, leading to the excessive release of dopamine in the nucleus accumbens. This creates a powerful sense of reward and reinforcement, encouraging repeated engagement in the addictive behavior despite adverse consequences.

Serotonin's role in addiction involves its regulatory effects on the reward circuits modulated by dopamine. It can modulate the impact of dopamine by influencing the reward system's sensitivity and response to stimuli. For instance, certain drugs of abuse can lead to a depletion of serotonin in the brain, altering mood and impulse control and making the dopaminergic reward pathway more compelling.

In summary, while dopamine directly drives the reinforcement and pleasure associated with rewards, serotonin plays a more modulatory role, influencing the valuation of rewards, impulsivity, and mood. Together, these neurotransmitters form a complex network of interactions that underpin the brain's response to rewarding stimuli, the development of addiction, and the challenges of overcoming addictive behaviors. Understanding these intricate mechanisms is crucial for developing effective treatments for addiction and related neuropsychiatric disorders.

4. Social media addiction


Next, let's explore the intricacies of how social media platforms exploit the same neural circuits in the brain implicated in substance addiction, such as those seen with heroin and cocaine use. At the Everyoung.com Research Centre, we have explored these mechanics extensively and reached a good understanding of the brain's reward system and how both substances and behaviors, including the use of social media, can co-opt it.

The Foundation: Brain's Reward System

At the heart of addiction—whether substance-related or behavioral—is the brain's reward system, primarily the mesolimbic dopamine system, which includes the ventral tegmental area (VTA) and the nucleus accumbens. Activation of this system releases dopamine, creating feelings of pleasure and reinforcing the behaviors leading to this activation. It has been explained clearly from the scientific aspect how addictive drugs artificially stimulate this pathway, reinforcing drug-taking behavior by increasing dopamine levels, often more rapidly and to a greater extent than natural rewards.

Social Media: A Behavioral Addiction

Social media, while not a chemical substance, can become addictive through mechanisms similar to drug addiction. This behavioral addiction capitalizes on the brain's tendency to repeat actions that lead to dopamine release in the reward pathway.

Intermittent Reinforcement. Social media platforms are designed to exploit the principle of intermittent reinforcement - rewards that are given at random intervals. This is similar to gambling and creates a powerful and compulsive engagement loop. Each scroll, like, or new post can be seen as pulling the lever on a slot machine, providing unpredictable rewards for likes, comments, or new content. This unpredictability is crucial to its addictive potential, as it keeps the brain in a state of anticipation, continually seeking the next reward.

Social Reward. Humans are inherently social creatures, and social interactions can activate the reward system. Social media platforms provide social rewards (likes, shares, comments) that trigger dopamine release (especially if one relates the interaction to one's personality), reinforcing the use of these platforms. Over time, the pursuit of these social rewards can become compulsive, mirroring the craving for drugs in substance addiction.

Normalization and sensitization. Just as with drug addiction, where repeated exposure to a drug leads to tolerance (requiring more of the drug to achieve the same effect) and sensitization (increased responsiveness to the drug), heavy social media use can alter the brain's reward system. Users may spend increasing amounts of time on social media to achieve the same level of satisfaction. They may become more sensitive to social rewards or punishments on these platforms.

Neuroplastic Changes. Repeated drug use leads to neuroplastic changes in the brain, rewiring the brain's reward circuitry to prioritize drug-related cues and activities over natural rewards. Similarly, heavy social media use can lead to neuroplastic changes prioritizing social media engagement. These changes can make abstaining from social media use difficult, as the brain has been conditioned to seek out the digital rewards it offers.

In conclusion, social media platforms, through their design and the nature of the rewards they offer, can become addictive by hijacking the same neural circuits that are exploited by addictive drugs. The intermittent, unpredictable nature of these rewards, combined with the intrinsic value we place on social interaction, creates a potent recipe for addiction. At the Everyoung.com Research Centre, we believe that understanding these parallels not only illuminates the addictive potential of social media but also underscores the importance of developing strategies to mitigate its impact on mental health and well-being.
 
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Randy
Friday, Apr 12, 2024

Great piece – I love the approach, the site is easy to browse and simple and serious topics such as addiction are discussed in a simple way!


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